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Ventilation a
response to mechanical, chemical, thermal and sensory changes Increased
vascular flow at alveolar site Surfactant peaks
at 35 weeks Characteristics rate 30-60, 60-70 first hour or two periodic 5-15
seconds not respiratory movement.
Longer periods greater than 20 seconds called apnea. This needs to be assessed Nasal breather if mucous need to clear
airway, can lead to respiratory distress Acrocyanosis Assess for
retractions, cyanosis, nasal flaring expiratory grunting, use of intercostal
muscles |
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Slide 3 |
Increased aortic
pressure Increased
systemic/decreased pulmonary pressure Closure of
foramen ovale functionally 1-2 hours after birth Closure of Ductus
arteriosus functionally within 15 hours after birth r/t prostaglandin E2 Measurement of HR Neonatal RBCs
have a life span of 80 to 100 days, 2/3 life span of adult RBC. In 1st days of life, hematocrit
may rise to 1 to 1 g/dL above fetal levels as a result of placental
transfusion, low oral fluid intake and diminished extracellular fluid
volume. By 1 week postnatally,
peripheral hemoglobin is comparable to fetal blood counts. The hgb level declines progressively over
the first 2 to 3 months of live. This
initial decline in hemoglobin creates a phenomenon know as physiologic anemia
of infancy. |
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Slide 6 |
Physiologic
Jaundice 1.
Increased amounts of bilirubin delivered to liver 2.
Defective hepatic uptake of bilirubin from the plasma 3. Defective conjugation of the bilirubin 4.
Defect in bilirubin excretion |
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Wt lost 5-10%
Shift ofintracellular water to extracellular space and insensible water loss Stool meconium
within j8-24 hours then transitional, thenbreast or bottle Urine within 48
hours. Voids in utero voids 2-6 times a
day day 1 and 2. Then 5-25 times a day volume of 24mL/kg per day |
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Slide 9 |
Immune system not
fully activated until after birth results in failure of newborns
inflammatory response result in failure to recognize, localize and destroy
invasive bacteria. S&S of
infection are subtle. Fever not a
reliable indicator of infection.
Hypothermia ia a more reliable sign of infection. Immunoglobulins-
IgG crosses placenta and gives passive acquired immunity IgM produced by
fetus usually response to blood group antigens, gram-negative enteric
organisms and some viruses in mom.Suggest exposure to TORCH infections |
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1st
awake, active, hungry, strong suck RR
rapid HR rapid and irregular Bowel
sounds absent Inactivity HR
RR decrease , sleep phase, no interest in sucking Bowel sound become audible 2nd
awake, alert. HR and RR increase,
apneic periods.Change color rapidly
Increase mucus responds to gagging, choking and regurgitating |
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Remind about
APGAR |
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Neuro resting
posture, square window, recoil, popliteal angle, scarf sign, heel-to-ear,
ankle dorsiflexion Physical skin,
lanugo, sole creases, areola, ear form and cartilage distribution, genitalia Plan nursing
interventions |
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Slide 21 |
Wt -
3405g or 78 Length 18-22 Head 32-37cm
12.5-14.5 Chest 30-35cm
12-14 Temp. 36.5-37
97.7-98.6 A hospital protocol for
reporting and intervention Skin
acrocyanosis, mottling, jaundice, erythema toxicum, milia, skin turgor,
vernix caseosa, forceps marks, telangiectatic nevi, mongolian spots,
port-wine stains, strawberry marks, Head
fontanelle, cephalhematoma, caput succedaneum, molding |
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Slide 28 |
Eyes
strabismus color treatment Mouth thrush
caused by candida albicans, Ears low set
r/t chromosomal abnormalities hearing
after first cry Respirations
already covered RR, and signs of respiratory difficulty Mention cry |
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Slide 29 |
CV already
talked about HR Apical pulse 120-160,
below 100 Abdomen
rounded, BS within 1 hour, not
scaphoid Cord Genitals Male
urinary meatus hypospadius (ventral
surface) criptorchidism Phimosis - female labia/maturation Anus patency meconium Extremities
digits, creases Hips ortolanis maneuver Back pilonidal
dimple, tuft of hair occult spina bifida Reflexes tonic
neck, moro, grasping, rooting sucking/swallow, babinski |